Shank3 Exons 14–16 Deletion in Glutamatergic Neurons Leads to Social and Repetitive Behavioral Deficits Associated With Increased Cortical Layer 2/3 Neuronal Excitability

Eitemau Tebyg

• GABA Neuronal Deletion of Shank3 Exons 14–16 in Mice Suppresses Striatal Excitatory Synaptic Input and Induces Social and Locomotor Abnormalities
  gan: Yoo, Taesun, et al.
  Cyhoeddwyd: (2018)
• Shank3-mutant mice lacking exon 9 show altered excitation/inhibition balance, enhanced rearing, and spatial memory deficit
  gan: Lee, Jiseok, et al.
  Cyhoeddwyd: (2015)
• Shank3 Mice Carrying the Human Q321R Mutation Display Enhanced Self-Grooming, Abnormal Electroencephalogram Patterns, and Suppressed Neuronal Excitability and Seizure Susceptibility
  gan: Yoo, Ye-Eun, et al.
  Cyhoeddwyd: (2019)
• Unexpected Compensatory Increase in Shank3 Transcripts in Shank3 Knock-Out Mice Having Partial Deletions of Exons
  gan: Jin, Chunmei, et al.
  Cyhoeddwyd: (2019)
• Shank2 Deletion in Parvalbumin Neurons Leads to Moderate Hyperactivity, Enhanced Self-Grooming and Suppressed Seizure Susceptibility in Mice
  gan: Lee, Seungjoon, et al.
  Cyhoeddwyd: (2018)