লোডিং...
Neuronal Deficiency of Presenilin 1 Inhibits Amyloid Plaque Formation and Corrects Hippocampal Long-Term Potentiation But Not a Cognitive Defect of Amyloid Precursor Protein [V717I] Transgenic Mice
In the brain of Alzheimer's disease (AD) patients, neurotoxic amyloid peptides accumulate and are deposited as senile plaques. A major therapeutic strategy aims to decrease production of amyloid peptides by inhibition of γ-secretase. Presenilins are polytopic transmembrane proteins that are ess...
সংরক্ষণ করুন:
| প্রকাশিত: | J Neurosci |
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| প্রধান লেখক: | , , , , , , , , , , , , , |
| বিন্যাস: | Artigo |
| ভাষা: | Inglês |
| প্রকাশিত: |
Society for Neuroscience
2002
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| বিষয়গুলি: | |
| অনলাইন ব্যবহার করুন: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6758348/ https://ncbi.nlm.nih.gov/pubmed/11978821 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.22-09-03445.2002 |
| ট্যাগগুলো: |
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