Neuronal Deficiency of Presenilin 1 Inhibits Amyloid Plaque Formation and Corrects Hippocampal Long-Term Potentiation But Not a Cognitive Defect of Amyloid Precursor Protein [V717I] Transgenic Mice

Lignende værker

• Aging Increased Amyloid Peptide and Caused Amyloid Plaques in Brain of Old APP/V717I Transgenic Mice by a Different Mechanism than Mutant Presenilin1
  af: Dewachter, Ilse, et al.
  Udgivet: (2000)
• Prominent Cerebral Amyloid Angiopathy in Transgenic Mice Overexpressing the London Mutant of Human APP in Neurons
  af: Van Dorpe, Jo, et al.
  Udgivet: (2000)
• Presenilin 2 deficiency causes a mild pulmonary phenotype and no changes in amyloid precursor protein processing but enhances the embryonic lethal phenotype of presenilin 1 deficiency
  af: Herreman, An, et al.
  Udgivet: (1999)
• α2-Macroglobulin- and Murinoglobulin-1- Deficient Mice : A Mouse Model for Acute Pancreatitis
  af: Umans, Lieve, et al.
  Udgivet: (1999)
• Expression of Human Apolipoprotein E4 in Neurons Causes Hyperphosphorylation of Protein Tau in the Brains of Transgenic Mice
  af: Tesseur, Ina, et al.
  Udgivet: (2000)