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Synaptic Targeting by Alzheimer's-Related Amyloid β Oligomers
The cognitive hallmark of early Alzheimer's disease (AD) is an extraordinary inability to form new memories. For many years, this dementia was attributed to nerve-cell death induced by deposits of fibrillar amyloid β (Aβ). A newer hypothesis has emerged, however, in which early memory loss is c...
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| Pubblicato in: | J Neurosci |
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| Autori principali: | , , , , , , , , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
Society for Neuroscience
2004
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6730194/ https://ncbi.nlm.nih.gov/pubmed/15537891 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.3432-04.2004 |
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