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Synaptic Targeting by Alzheimer's-Related Amyloid β Oligomers

The cognitive hallmark of early Alzheimer's disease (AD) is an extraordinary inability to form new memories. For many years, this dementia was attributed to nerve-cell death induced by deposits of fibrillar amyloid β (Aβ). A newer hypothesis has emerged, however, in which early memory loss is c...

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Bibliographic Details
Published in:J Neurosci
Main Authors: Lacor, Pascale N., Buniel, Maria C., Chang, Lei, Fernandez, Sara J., Gong, Yuesong, Viola, Kirsten L., Lambert, Mary P., Velasco, Pauline T., Bigio, Eileen H., Finch, Caleb E., Krafft, Grant A., Klein, William L.
Format: Artigo
Language:Inglês
Published: Society for Neuroscience 2004
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Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC6730194/
https://ncbi.nlm.nih.gov/pubmed/15537891
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.3432-04.2004
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