Synaptic Targeting by Alzheimer's-Related Amyloid β Oligomers

The cognitive hallmark of early Alzheimer's disease (AD) is an extraordinary inability to form new memories. For many years, this dementia was attributed to nerve-cell death induced by deposits of fibrillar amyloid β (Aβ). A newer hypothesis has emerged, however, in which early memory loss is c...

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Detalhes bibliográficos
Publicado no:J Neurosci
Main Authors: Lacor, Pascale N., Buniel, Maria C., Chang, Lei, Fernandez, Sara J., Gong, Yuesong, Viola, Kirsten L., Lambert, Mary P., Velasco, Pauline T., Bigio, Eileen H., Finch, Caleb E., Krafft, Grant A., Klein, William L.
Formato: Artigo
Idioma:Inglês
Publicado em: Society for Neuroscience 2004
Assuntos:
Neurobiology of Disease
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6730194/
https://ncbi.nlm.nih.gov/pubmed/15537891
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.3432-04.2004
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