Synaptic Targeting by Alzheimer's-Related Amyloid β Oligomers

The cognitive hallmark of early Alzheimer's disease (AD) is an extraordinary inability to form new memories. For many years, this dementia was attributed to nerve-cell death induced by deposits of fibrillar amyloid β (Aβ). A newer hypothesis has emerged, however, in which early memory loss is c...

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Bibliografiska uppgifter
I publikationen:J Neurosci
Huvudupphovsmän: Lacor, Pascale N., Buniel, Maria C., Chang, Lei, Fernandez, Sara J., Gong, Yuesong, Viola, Kirsten L., Lambert, Mary P., Velasco, Pauline T., Bigio, Eileen H., Finch, Caleb E., Krafft, Grant A., Klein, William L.
Materialtyp: Artigo
Språk:Inglês
Publicerad: Society for Neuroscience 2004
Ämnen:
Neurobiology of Disease
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC6730194/
https://ncbi.nlm.nih.gov/pubmed/15537891
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.3432-04.2004
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