Synaptic Targeting by Alzheimer's-Related Amyloid β Oligomers

The cognitive hallmark of early Alzheimer's disease (AD) is an extraordinary inability to form new memories. For many years, this dementia was attributed to nerve-cell death induced by deposits of fibrillar amyloid β (Aβ). A newer hypothesis has emerged, however, in which early memory loss is c...

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Bibliografski detalji
Izdano u:J Neurosci
Glavni autori: Lacor, Pascale N., Buniel, Maria C., Chang, Lei, Fernandez, Sara J., Gong, Yuesong, Viola, Kirsten L., Lambert, Mary P., Velasco, Pauline T., Bigio, Eileen H., Finch, Caleb E., Krafft, Grant A., Klein, William L.
Format: Artigo
Jezik:Inglês
Izdano: Society for Neuroscience 2004
Teme:
Neurobiology of Disease
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC6730194/
https://ncbi.nlm.nih.gov/pubmed/15537891
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.3432-04.2004
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