Synaptic Targeting by Alzheimer's-Related Amyloid β Oligomers

The cognitive hallmark of early Alzheimer's disease (AD) is an extraordinary inability to form new memories. For many years, this dementia was attributed to nerve-cell death induced by deposits of fibrillar amyloid β (Aβ). A newer hypothesis has emerged, however, in which early memory loss is c...

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Pubblicato in:J Neurosci
Autori principali: Lacor, Pascale N., Buniel, Maria C., Chang, Lei, Fernandez, Sara J., Gong, Yuesong, Viola, Kirsten L., Lambert, Mary P., Velasco, Pauline T., Bigio, Eileen H., Finch, Caleb E., Krafft, Grant A., Klein, William L.
Natura: Artigo
Lingua:Inglês
Pubblicazione: Society for Neuroscience 2004
Soggetti:
Neurobiology of Disease
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC6730194/
https://ncbi.nlm.nih.gov/pubmed/15537891
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.3432-04.2004
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