Experimental colitis in IL‐10‐deficient mice ameliorates in the absence of PTPN22

Interleukin (IL)‐10 plays a key role in controlling intestinal inflammation. IL‐10‐deficient mice and patients with mutations in IL‐10 or its receptor, IL‐10R, show increased susceptibility to inflammatory bowel diseases (IBD). Protein tyrosine phosphatase, non‐receptor type 22 (PTPN22) controls imm...

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Bibliografiske detaljer
Udgivet i:Clin Exp Immunol
Main Authors: Jofra, T., Galvani, G., Cosorich, I., De Giorgi, L., Annoni, A., Vecchione, A., Sorini, C., Falcone, M., Fousteri, G.
Format: Artigo
Sprog:Inglês
Udgivet: John Wiley and Sons Inc. 2019
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Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC6693971/
https://ncbi.nlm.nih.gov/pubmed/31194881
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/cei.13339
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