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Experimental colitis in IL‐10‐deficient mice ameliorates in the absence of PTPN22
Interleukin (IL)‐10 plays a key role in controlling intestinal inflammation. IL‐10‐deficient mice and patients with mutations in IL‐10 or its receptor, IL‐10R, show increased susceptibility to inflammatory bowel diseases (IBD). Protein tyrosine phosphatase, non‐receptor type 22 (PTPN22) controls imm...
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| Pubblicato in: | Clin Exp Immunol |
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| Autori principali: | , , , , , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
John Wiley and Sons Inc.
2019
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6693971/ https://ncbi.nlm.nih.gov/pubmed/31194881 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/cei.13339 |
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