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Experimental colitis in IL‐10‐deficient mice ameliorates in the absence of PTPN22
Interleukin (IL)‐10 plays a key role in controlling intestinal inflammation. IL‐10‐deficient mice and patients with mutations in IL‐10 or its receptor, IL‐10R, show increased susceptibility to inflammatory bowel diseases (IBD). Protein tyrosine phosphatase, non‐receptor type 22 (PTPN22) controls imm...
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| Gepubliceerd in: | Clin Exp Immunol |
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| Hoofdauteurs: | , , , , , , , , |
| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
John Wiley and Sons Inc.
2019
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6693971/ https://ncbi.nlm.nih.gov/pubmed/31194881 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/cei.13339 |
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