Experimental colitis in IL‐10‐deficient mice ameliorates in the absence of PTPN22

Interleukin (IL)‐10 plays a key role in controlling intestinal inflammation. IL‐10‐deficient mice and patients with mutations in IL‐10 or its receptor, IL‐10R, show increased susceptibility to inflammatory bowel diseases (IBD). Protein tyrosine phosphatase, non‐receptor type 22 (PTPN22) controls imm...

詳細記述

保存先:
書誌詳細
出版年:Clin Exp Immunol
主要な著者: Jofra, T., Galvani, G., Cosorich, I., De Giorgi, L., Annoni, A., Vecchione, A., Sorini, C., Falcone, M., Fousteri, G.
フォーマット: Artigo
言語:Inglês
出版事項: John Wiley and Sons Inc. 2019
主題:
Editors’ Choice
オンライン･アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC6693971/
https://ncbi.nlm.nih.gov/pubmed/31194881
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/cei.13339
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