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Experimental colitis in IL‐10‐deficient mice ameliorates in the absence of PTPN22
Interleukin (IL)‐10 plays a key role in controlling intestinal inflammation. IL‐10‐deficient mice and patients with mutations in IL‐10 or its receptor, IL‐10R, show increased susceptibility to inflammatory bowel diseases (IBD). Protein tyrosine phosphatase, non‐receptor type 22 (PTPN22) controls imm...
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| Publicado no: | Clin Exp Immunol |
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| Main Authors: | , , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
John Wiley and Sons Inc.
2019
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6693971/ https://ncbi.nlm.nih.gov/pubmed/31194881 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/cei.13339 |
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