Cellular Prion Protein as a Receptor of Toxic Amyloid-β42 Oligomers Is Important for Alzheimer’s Disease

The pathological features of Alzheimer’s disease (AD) include senile plaques induced by amyloid-β (Aβ) protein deposits, neurofibrillary tangles formed by aggregates of hyperphosphorylated tau proteins and neuronal cell loss in specific position within the brain. Recent observations have suggested t...

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Bibliographic Details
Published in:Front Cell Neurosci
Main Authors: Zhang, Yuan, Zhao, Yanfang, Zhang, Lei, Yu, Wanpeng, Wang, Yu, Chang, Wenguang
Format: Artigo
Language:Inglês
Published: Frontiers Media S.A. 2019
Subjects:
Neuroscience
Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC6682659/
https://ncbi.nlm.nih.gov/pubmed/31417361
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fncel.2019.00339
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