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Cross-Seeding Fibrillation of Q/N-Rich Proteins Offers New Pathomechanism of Polyglutamine Diseases

A pathological hallmark of the Huntington's disease (HD) is intracellular inclusions containing a huntingtin (Htt) protein with an elongated polyglutamine tract. Aggregation of mutant Htt causes abnormal protein–protein interactions, and the functional dysregulation of aggregate-interacting pro...

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Bibliografske podrobnosti
izdano v:J Neurosci
Main Authors: Furukawa, Yoshiaki, Kaneko, Kumi, Matsumoto, Gen, Kurosawa, Masaru, Nukina, Nobuyuki
Format: Artigo
Jezik:Inglês
Izdano: Society for Neuroscience 2009
Teme:
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC6665470/
https://ncbi.nlm.nih.gov/pubmed/19386911
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.0783-09.2009
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