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Kidney injury is independent of endothelial HIF-1α
Hypoxia-inducible transcription factors (HIFs) control cellular adaptation to low oxygen. In the kidney, activation of HIF is beneficial during injury; however, the specific contribution of HIF-1α in renal endothelial cells (EC) remains elusive. Since EC display tissue-specific heterogeneity, we inv...
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| Publicat a: | J Mol Med (Berl) |
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| Autors principals: | , , , , , , , , , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2015
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6592817/ https://ncbi.nlm.nih.gov/pubmed/25754172 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s00109-015-1264-4 |
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