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Kidney injury is independent of endothelial HIF-1α

Hypoxia-inducible transcription factors (HIFs) control cellular adaptation to low oxygen. In the kidney, activation of HIF is beneficial during injury; however, the specific contribution of HIF-1α in renal endothelial cells (EC) remains elusive. Since EC display tissue-specific heterogeneity, we inv...

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Detalhes bibliográficos
Publicado no:J Mol Med (Berl)
Main Authors: Kalucka, Joanna, Schley, Gunnar, Georgescu, Adela, Klanke, Bernd, Rössler, Susanne, Baumgartl, Jasmin, Velden, Joachim, Amann, Kerstin, Willam, Carsten, Johnson, Randall S., Eckardt, Kai-Uwe, Weidemann, Alexander
Formato: Artigo
Idioma:Inglês
Publicado em: 2015
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6592817/
https://ncbi.nlm.nih.gov/pubmed/25754172
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s00109-015-1264-4
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