טוען...
Dual gain and loss of cullin 3 function mediates familial hyperkalemic hypertension
Familial hyperkalemic hypertension is caused by mutations in with-no-lysine kinases (WNKs) or in proteins that mediate their degradation, kelch-like 3 (KLHL3) and cullin 3 (CUL3). Although the mechanisms by which WNK and KLHL3 mutations cause the disease are now clear, the effects of the disease-cau...
שמור ב:
| הוצא לאור ב: | Am J Physiol Renal Physiol |
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| Main Authors: | , , , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
American Physiological Society
2018
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6230741/ https://ncbi.nlm.nih.gov/pubmed/29897280 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00602.2017 |
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