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Reviving oncogenic addiction to MET bypassed by BRAF (G469A) mutation
Cancer clonal evolution is based on accrual of driving genetic alterations that are expected to cooperate and progressively increase malignancy. Little is known on whether any genetic alteration can hinder the oncogenic function of a coexisting alteration, so that therapeutic targeting of the one ca...
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| Publicado en: | Proc Natl Acad Sci U S A |
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| Autores principales: | , , , |
| Formato: | Artigo |
| Lenguaje: | Inglês |
| Publicado: |
National Academy of Sciences
2018
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| Materias: | |
| Acceso en línea: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6176587/ https://ncbi.nlm.nih.gov/pubmed/30224486 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1721147115 |
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