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Reviving oncogenic addiction to MET bypassed by BRAF (G469A) mutation
Cancer clonal evolution is based on accrual of driving genetic alterations that are expected to cooperate and progressively increase malignancy. Little is known on whether any genetic alteration can hinder the oncogenic function of a coexisting alteration, so that therapeutic targeting of the one ca...
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| Publicado no: | Proc Natl Acad Sci U S A |
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| Main Authors: | , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
National Academy of Sciences
2018
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6176587/ https://ncbi.nlm.nih.gov/pubmed/30224486 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1721147115 |
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