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MIG-6 suppresses endometrial epithelial cell proliferation by inhibiting phospho-AKT
BACKGROUND: Aberrant hyperactivation of epithelial proliferation, AKT signaling, and association with unopposed estrogen (E2) exposure is the most common endometrial cancer dysfunction. In the normal uterus, progesterone (P4) inhibits proliferation by coordinating stromal-epithelial cross-talk, whic...
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| 出版年: | BMC Cancer |
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| 主要な著者: | , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
BioMed Central
2018
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5975686/ https://ncbi.nlm.nih.gov/pubmed/29843645 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12885-018-4502-7 |
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