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Leukotriene D(4) and prostaglandin E(2) signals synergize and potentiate vascular inflammation in a mast cell–dependent manner through cysteinyl leukotriene receptor 1 and E-prostanoid receptor 3
BACKGROUND: Although arachidonic acid metabolites, cysteinyl leukotrienes (cys-LTs; leukotriene [LT] C(4), LTD(4), and LTE(4)), and prostaglandin (PG) E(2) are generated at the site of inflammation, it is not known whether crosstalk exists between these 2 classes of inflammatory mediators. OBJECTIVE...
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| Publicado no: | J Allergy Clin Immunol |
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| Main Authors: | , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2015
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5839097/ https://ncbi.nlm.nih.gov/pubmed/26255103 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.jaci.2015.06.030 |
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