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Advanced glycation end products impair NLRP3 inflammasome–mediated innate immune responses in macrophages
Advanced glycation end products (AGEs) are adducts formed on proteins by glycation with reducing sugars, such as glucose, and tend to form and accumulate under hyperglycemic conditions. AGE accumulation alters protein function and has been implicated in the pathogenesis of many degenerative diseases...
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| 出版年: | J Biol Chem |
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| 主要な著者: | , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Society for Biochemistry and Molecular Biology
2017
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5733583/ https://ncbi.nlm.nih.gov/pubmed/29051224 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M117.806307 |
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