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Angptl4 does not control hyperglucagonemia or α-cell hyperplasia following glucagon receptor inhibition

Genetic disruption or pharmacologic inhibition of glucagon signaling effectively lowers blood glucose but results in compensatory glucagon hypersecretion involving expansion of pancreatic α-cell mass. Ben-Zvi et al. recently reported that angiopoietin-like protein 4 (Angptl4) links glucagon receptor...

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Detalles Bibliográficos
Publicado en:Proc Natl Acad Sci U S A
Main Authors: Okamoto, Haruka, Cavino, Katie, Na, Erqian, Krumm, Elizabeth, Kim, Steven, Stevis, Panayiotis E., Harp, Joyce, Murphy, Andrew J., Yancopoulos, George D., Gromada, Jesper
Formato: Artigo
Idioma:Inglês
Publicado: National Academy of Sciences 2017
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Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC5347591/
https://ncbi.nlm.nih.gov/pubmed/28143927
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1620989114
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