Angptl4 does not control hyperglucagonemia or α-cell hyperplasia following glucagon receptor inhibition

Genetic disruption or pharmacologic inhibition of glucagon signaling effectively lowers blood glucose but results in compensatory glucagon hypersecretion involving expansion of pancreatic α-cell mass. Ben-Zvi et al. recently reported that angiopoietin-like protein 4 (Angptl4) links glucagon receptor...

Popoln opis

Shranjeno v:
Bibliografske podrobnosti
izdano v:Proc Natl Acad Sci U S A
Main Authors: Okamoto, Haruka, Cavino, Katie, Na, Erqian, Krumm, Elizabeth, Kim, Steven, Stevis, Panayiotis E., Harp, Joyce, Murphy, Andrew J., Yancopoulos, George D., Gromada, Jesper
Format: Artigo
Jezik:Inglês
Izdano: National Academy of Sciences 2017
Teme:
Biological Sciences
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC5347591/
https://ncbi.nlm.nih.gov/pubmed/28143927
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1620989114
Oznake: Označite
Brez oznak, prvi označite!

Podobne knjige/članki