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Toll-like receptor 9 suppresses lupus disease in Fas-sufficient MRL Mice

Genetic deficiency in TLR9 accelerates pathogenesis in the spontaneous polygenic MRL.Fas(lpr) murine model of systemic lupus erythematosus, despite the absence of anti-nucleosome autoantibodies. However, it could be argued that this result was dependent on Fas-deficiency rather than lupus-promoting...

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Detaylı Bibliyografya
Yayımlandı:PLoS One
Asıl Yazarlar: Nickerson, Kevin M., Wang, Yujuan, Bastacky, Sheldon, Shlomchik, Mark J.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Public Library of Science 2017
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC5344451/
https://ncbi.nlm.nih.gov/pubmed/28278279
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0173471
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