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Murine lupus is neutrophil elastase-independent in the MRL.Fas(lpr) model
Loss of tolerance to nuclear antigens and multisystem tissue destruction is a hallmark of systemic lupus erythematosus (SLE). Although the source of autoantigen in lupus remains elusive, a compelling hypothetical source is dead cell debris that drives autoimmune activation. Prior reports suggest tha...
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| Gepubliceerd in: | PLoS One |
|---|---|
| Hoofdauteurs: | , , , , , |
| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
Public Library of Science
2020
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7122749/ https://ncbi.nlm.nih.gov/pubmed/32243431 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0226396 |
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