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Tdp-43 cryptic exons are highly variable between cell types
BACKGROUND: TDP-43 proteinopathy is a prominent pathological feature that occurs in a number of human diseases including amyotrophic lateral sclerosis (ALS), frontotemporal dementia (FTD), and inclusion body myositis (IBM). Our recent finding that TDP-43 represses nonconserved cryptic exons led us t...
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| Udgivet i: | Mol Neurodegener |
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| Main Authors: | , , , , , , , , , |
| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
BioMed Central
2017
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5289002/ https://ncbi.nlm.nih.gov/pubmed/28153034 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s13024-016-0144-x |
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