A20 Ameliorates Intracerebral Hemorrhage–Induced Inflammatory Injury by Regulating TRAF6 Polyubiquitination

Reducing excessive inflammation is beneficial for the recovery from intracerebral hemorrhage (ICH). Here, the roles and mechanisms of A20 (TNFAIP3), an important endogenous anti-inflammatory factor, are examined in ICH. A20 expression in the PBMCs of ICH patients and an ICH mouse model was detected,...

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Vydáno v:J Immunol
Hlavní autoři: Meng, Zhaoyou, Zhao, Ting, Zhou, Kai, Zhong, Qi, Wang, Yanchun, Xiong, Xiaoyi, Wang, Faxiang, Yang, Yuanrui, Zhu, Wenyao, Liu, Juan, Liao, Maofan, Wu, Lirong, Duan, Chunmei, Li, Jie, Gong, Qiuwen, Liu, Liang, Xiong, Ao, Yang, Meihua, Wang, Jian, Yang, Qingwu
Médium: Artigo
Jazyk:Inglês
Vydáno: AAI 2017
Témata:
Innate Immunity and Inflammation
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC5220121/
https://ncbi.nlm.nih.gov/pubmed/27986908
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1600334
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