A20 Ameliorates Intracerebral Hemorrhage–Induced Inflammatory Injury by Regulating TRAF6 Polyubiquitination

Reducing excessive inflammation is beneficial for the recovery from intracerebral hemorrhage (ICH). Here, the roles and mechanisms of A20 (TNFAIP3), an important endogenous anti-inflammatory factor, are examined in ICH. A20 expression in the PBMCs of ICH patients and an ICH mouse model was detected,...

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Dettagli Bibliografici
Pubblicato in:J Immunol
Autori principali: Meng, Zhaoyou, Zhao, Ting, Zhou, Kai, Zhong, Qi, Wang, Yanchun, Xiong, Xiaoyi, Wang, Faxiang, Yang, Yuanrui, Zhu, Wenyao, Liu, Juan, Liao, Maofan, Wu, Lirong, Duan, Chunmei, Li, Jie, Gong, Qiuwen, Liu, Liang, Xiong, Ao, Yang, Meihua, Wang, Jian, Yang, Qingwu
Natura: Artigo
Lingua:Inglês
Pubblicazione: AAI 2017
Soggetti:
Innate Immunity and Inflammation
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC5220121/
https://ncbi.nlm.nih.gov/pubmed/27986908
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1600334
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