A20 Ameliorates Intracerebral Hemorrhage–Induced Inflammatory Injury by Regulating TRAF6 Polyubiquitination

Reducing excessive inflammation is beneficial for the recovery from intracerebral hemorrhage (ICH). Here, the roles and mechanisms of A20 (TNFAIP3), an important endogenous anti-inflammatory factor, are examined in ICH. A20 expression in the PBMCs of ICH patients and an ICH mouse model was detected,...

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Detalles Bibliográficos
Publicado en:J Immunol
Main Authors: Meng, Zhaoyou, Zhao, Ting, Zhou, Kai, Zhong, Qi, Wang, Yanchun, Xiong, Xiaoyi, Wang, Faxiang, Yang, Yuanrui, Zhu, Wenyao, Liu, Juan, Liao, Maofan, Wu, Lirong, Duan, Chunmei, Li, Jie, Gong, Qiuwen, Liu, Liang, Xiong, Ao, Yang, Meihua, Wang, Jian, Yang, Qingwu
Formato: Artigo
Idioma:Inglês
Publicado: AAI 2017
Assuntos:
Innate Immunity and Inflammation
Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC5220121/
https://ncbi.nlm.nih.gov/pubmed/27986908
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1600334
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