Paradoxical leanness in the imprinting-centre deletion mouse model for Prader–Willi syndrome

Prader–Willi syndrome (PWS), a neurodevelopmental disorder caused by loss of paternal gene expression from 15q11–q13, is characterised by growth retardation, hyperphagia and obesity. However, as single gene mutation mouse models for this condition display an incomplete spectrum of the PWS phenotype,...

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Shranjeno v:
Bibliografske podrobnosti
izdano v:J Endocrinol
Main Authors: Golding, David M, Rees, Daniel J, Davies, Jennifer R, Relkovic, Dinko, Furby, Hannah V, Guschina, Irina A, Hopkins, Anna L, Davies, Jeffrey S, Resnick, James L, Isles, Anthony R, Wells, Timothy
Format: Artigo
Jezik:Inglês
Izdano: Bioscientifica Ltd 2016
Teme:
Research
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC5118940/
https://ncbi.nlm.nih.gov/pubmed/27799465
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1530/JOE-16-0367
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