Paradoxical leanness in the imprinting-centre deletion mouse model for Prader–Willi syndrome

Prader–Willi syndrome (PWS), a neurodevelopmental disorder caused by loss of paternal gene expression from 15q11–q13, is characterised by growth retardation, hyperphagia and obesity. However, as single gene mutation mouse models for this condition display an incomplete spectrum of the PWS phenotype,...

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Bibliografische gegevens
Gepubliceerd in:J Endocrinol
Hoofdauteurs: Golding, David M, Rees, Daniel J, Davies, Jennifer R, Relkovic, Dinko, Furby, Hannah V, Guschina, Irina A, Hopkins, Anna L, Davies, Jeffrey S, Resnick, James L, Isles, Anthony R, Wells, Timothy
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: Bioscientifica Ltd 2016
Onderwerpen:
Research
Online toegang:https://ncbi.nlm.nih.gov/pmc/articles/PMC5118940/
https://ncbi.nlm.nih.gov/pubmed/27799465
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1530/JOE-16-0367
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