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Targeting Inhibitor of Apoptosis Proteins Protects from Bleomycin-Induced Lung Fibrosis
Accumulation of apoptosis-resistant fibroblasts is a hallmark of pulmonary fibrosis. We hypothesized that disruption of inhibitor of apoptosis protein (IAP) family proteins would limit lung fibrosis. We first show that transforming growth factor-β1 and bleomycin increase X-linked IAP (XIAP) and cell...
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| 出版年: | Am J Respir Cell Mol Biol |
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| 主要な著者: | , , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Thoracic Society
2016
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4821054/ https://ncbi.nlm.nih.gov/pubmed/26378893 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1165/rcmb.2015-0148OC |
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