IL-17A deficiency mitigates bleomycin-induced complement activation during lung fibrosis

Interleukin 17A (IL-17A) and complement (C′) activation have each been implicated in the pathogenesis of idiopathic pulmonary fibrosis (IPF). We have reported that IL-17A induces epithelial injury via TGF-β in murine bronchiolitis obliterans; that TGF-β and the C′ cascade present signaling interacti...

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Detalhes bibliográficos
Publicado no:FASEB J
Main Authors: Cipolla, Ellyse, Fisher, Amanda J., Gu, Hongmei, Mickler, Elizabeth A., Agarwal, Manisha, Wilke, Carol A., Kim, Kevin K., Moore, Bethany B., Vittal, Ragini
Formato: Artigo
Idioma:Inglês
Publicado em: Federation of American Societies for Experimental Biology 2017
Assuntos:
Research
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5690386/
https://ncbi.nlm.nih.gov/pubmed/28821630
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1096/fj.201700289R
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