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Early and delayed intervention with Rapamycin prevents NNK-induced lung adenocarcinoma in A/J mice
In tobacco-associated lung cancers, the protein kinase B/mammalian target of rapamycin (Akt/mTOR) pathway frequently is activated by nicotine and its metabolite 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK). The aim of the present study was to examine the effects of early or late intervention...
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| 發表在: | Oncol Rep |
|---|---|
| Main Authors: | , , , , , , , , , , |
| 格式: | Artigo |
| 語言: | Inglês |
| 出版: |
D.A. Spandidos
2015
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| 主題: | |
| 在線閱讀: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4735698/ https://ncbi.nlm.nih.gov/pubmed/26397133 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3892/or.2015.4277 |
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