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β(3)-Adrenoceptor activation relieves oxidative inhibition of the cardiac Na(+)-K(+) pump in hyperglycemia induced by insulin receptor blockade
Dysregulated nitric oxide (NO)- and superoxide (O(2)(·−))-dependent signaling contributes to the pathobiology of diabetes-induced cardiovascular complications. We examined if stimulation of β(3)-adrenergic receptors (β(3)-ARs), coupled to endothelial NO synthase (eNOS) activation, relieves oxidative...
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| Gepubliceerd in: | Am J Physiol Cell Physiol |
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| Hoofdauteurs: | , , , , , , |
| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
American Physiological Society
2015
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4556897/ https://ncbi.nlm.nih.gov/pubmed/26063704 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpcell.00071.2015 |
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