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Synaptotagmins interact with APP and promote Aβ generation
BACKGROUND: Accumulation of the β-amyloid peptide (Aβ) is a major pathological hallmark of Alzheimer’s disease (AD). Recent studies have shown that synaptic Aβ toxicity may directly impair synaptic function. However, proteins regulating Aβ generation at the synapse have not been characterized. Here,...
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| Publicat a: | Mol Neurodegener |
|---|---|
| Autors principals: | , , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
BioMed Central
2015
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4511450/ https://ncbi.nlm.nih.gov/pubmed/26202512 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s13024-015-0028-5 |
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