Relationship between decay accelerating factor deficiency, diminished acetylcholinesterase activity, and defective terminal complement pathway restriction in paroxysmal nocturnal hemoglobinuria erythrocytes.

Podobne zapisy

• Amelioration of lytic abnormalities of paroxysmal nocturnal hemoglobinuria with decay-accelerating factor.
  od: Medof, M E, i wsp.
  Wydane: (1985)
• Characterization of the complement sensitivity of paroxysmal nocturnal hemoglobinuria erythrocytes.
  od: Parker, C J, i wsp.
  Wydane: (1985)
• Affected erythrocytes of patients with paroxysmal nocturnal hemoglobinuria are deficient in the complement regulatory protein, decay accelerating factor.
  od: Nicholson-Weller, A, i wsp.
  Wydane: (1983)
• Normal polymorphic variations and transcription of the decay accelerating factor gene in paroxysmal nocturnal hemoglobinuria cells.
  od: Stafford, H A, i wsp.
  Wydane: (1988)
• Synthesis of aberrant decay-accelerating factor proteins by affected paroxysmal nocturnal hemoglobinuria leukocytes.
  od: Carothers, D J, i wsp.
  Wydane: (1990)