Deletion of Ribosomal S6 Kinases Does Not Attenuate Pathological, Physiological, or Insulin-Like Growth Factor 1 Receptor-Phosphoinositide 3-Kinase-Induced Cardiac Hypertrophy

Ribosomal S6 kinases (S6Ks) have been depicted as critical effectors downstream of growth factor pathways, which play an important role in the regulation of protein synthesis by phosphorylating the ribosomal protein, S6. The goal of this study was to determine whether S6Ks regulate heart size, are c...

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Autors principals: McMullen, Julie R., Shioi, Tetsuo, Zhang, Li, Tarnavski, Oleg, Sherwood, Megan C., Dorfman, Adam L., Longnus, Sarah, Pende, Mario, Martin, Kathleen A., Blenis, John, Thomas, George, Izumo, Seigo
Format: Artigo
Idioma:Inglês
Publicat: American Society for Microbiology 2004
Matèries:
Mammalian Genetic Models with Minimal or Complex Phenotypes
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC434247/
https://ncbi.nlm.nih.gov/pubmed/15226426
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/MCB.24.14.6231-6240.2004
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