Deletion of Ribosomal S6 Kinases Does Not Attenuate Pathological, Physiological, or Insulin-Like Growth Factor 1 Receptor-Phosphoinositide 3-Kinase-Induced Cardiac Hypertrophy

Ribosomal S6 kinases (S6Ks) have been depicted as critical effectors downstream of growth factor pathways, which play an important role in the regulation of protein synthesis by phosphorylating the ribosomal protein, S6. The goal of this study was to determine whether S6Ks regulate heart size, are c...

Descripción completa

Guardado en:
Detalles Bibliográficos
Autores principales: McMullen, Julie R., Shioi, Tetsuo, Zhang, Li, Tarnavski, Oleg, Sherwood, Megan C., Dorfman, Adam L., Longnus, Sarah, Pende, Mario, Martin, Kathleen A., Blenis, John, Thomas, George, Izumo, Seigo
Formato: Artigo
Lenguaje:Inglês
Publicado: American Society for Microbiology 2004
Materias:
Mammalian Genetic Models with Minimal or Complex Phenotypes
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC434247/
https://ncbi.nlm.nih.gov/pubmed/15226426
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/MCB.24.14.6231-6240.2004
Etiquetas: Agregar Etiqueta
Sin Etiquetas, Sea el primero en etiquetar este registro!

Ejemplares similares