Class I(A) Phosphoinositide 3-Kinase Regulates Heart Size and Physiological Cardiac Hypertrophy

Eitemau Tebyg

• Phosphoinositide 3-kinase(p110α) plays a critical role for the induction of physiological, but not pathological, cardiac hypertrophy
  gan: McMullen, Julie R., et al.
  Cyhoeddwyd: (2003)
• Deletion of Ribosomal S6 Kinases Does Not Attenuate Pathological, Physiological, or Insulin-Like Growth Factor 1 Receptor-Phosphoinositide 3-Kinase-Induced Cardiac Hypertrophy
  gan: McMullen, Julie R., et al.
  Cyhoeddwyd: (2004)
• Correction for McMullen et al., “Deletion of Ribosomal S6 Kinases Does Not Attenuate Pathological, Physiological, or Insulin-Like Growth Factor 1 Receptor–Phosphoinositide 3-Kinase-Induced Cardiac Hypertrophy”
  gan: McMullen, Julie R., et al.
  Cyhoeddwyd: (2021)
• Transcription Factor GATA4 Is Activated but Not Required for Insulin-like Growth Factor 1 (IGF1)-induced Cardiac Hypertrophy
  gan: Bisping, Egbert, et al.
  Cyhoeddwyd: (2012)
• Modulation of epithelial neoplasia and lymphoid hyperplasia in PTEN(+/-) mice by the p85 regulatory subunits of phosphoinositide 3-kinase
  gan: Luo, Ji, et al.
  Cyhoeddwyd: (2005)