Phosphoinositide 3-kinase(p110α) plays a critical role for the induction of physiological, but not pathological, cardiac hypertrophy

Antzeko izenburuak

• Deletion of Ribosomal S6 Kinases Does Not Attenuate Pathological, Physiological, or Insulin-Like Growth Factor 1 Receptor-Phosphoinositide 3-Kinase-Induced Cardiac Hypertrophy
  nork: McMullen, Julie R., et al.
  Argitaratua: (2004)
• Correction for McMullen et al., “Deletion of Ribosomal S6 Kinases Does Not Attenuate Pathological, Physiological, or Insulin-Like Growth Factor 1 Receptor–Phosphoinositide 3-Kinase-Induced Cardiac Hypertrophy”
  nork: McMullen, Julie R., et al.
  Argitaratua: (2021)
• Class I(A) Phosphoinositide 3-Kinase Regulates Heart Size and Physiological Cardiac Hypertrophy
  nork: Luo, Ji, et al.
  Argitaratua: (2005)
• Phosphoinositide 3-kinase Akt signaling pathway interacts with protein kinase Cβ2 in the regulation of physiologic developmental hypertrophy and heart function
  nork: Rigor, Debra L., et al.
  Argitaratua: (2009)
• Protective effects of exercise and phosphoinositide 3-kinase(p110α) signaling in dilated and hypertrophic cardiomyopathy
  nork: McMullen, Julie R., et al.
  Argitaratua: (2007)