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Erythropoietin prevents endothelial dysfunction in GTP-cyclohydrolase I-deficient hph1 mice
In the present study, we used the mutant hph-1 mouse model, that has deficiency in GTP-cyclohydrolase I (GTPCH I) activity, to test the hypothesis that erythropoietin (EPO) protects aortic wall from oxidative stress induced by uncoupling of endothelial nitric oxide synthase (eNOS). Both GTPCH I acti...
Shranjeno v:
| izdano v: | J Cardiovasc Pharmacol |
|---|---|
| Main Authors: | , , |
| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
2014
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| Teme: | |
| Online dostop: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4261745/ https://ncbi.nlm.nih.gov/pubmed/25490417 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1097/FJC.0000000000000145 |
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