Mitochondrial-nuclear genome interactions in nonalcoholic fatty liver disease in mice

Lignende værker

• The methyl donor S-adenosylmethionine prevents liver hypoxia and dysregulation of mitochondrial bioenergetic function in a rat model of alcohol-induced fatty liver disease
  af: King, Adrienne L., et al.
  Udgivet: (2016)
• Involvement of the mitochondrial permeability transition pore in chronic ethanol-mediated liver injury in mice
  af: King, Adrienne L., et al.
  Udgivet: (2013)
• Mitochondrial genetics regulate nuclear gene expression through metabolites
  af: Fetterman, Jessica L., et al.
  Udgivet: (2019)
• Chronic ethanol consumption enhances sensitivity to Ca(2+)-mediated opening of the mitochondrial permeability transition pore and increases cyclophilin D in liver
  af: King, Adrienne L., et al.
  Udgivet: (2010)
• Ethanol and tobacco smoke increase hepatic steatosis and hypoxia in the hypercholesterolemic apoE(-/-) mouse: implications for a “multi-hit” hypothesis of fatty liver disease
  af: Bailey, Shannon M., et al.
  Udgivet: (2009)