Transgenic Mice Overexpressing Amyloid Precursor Protein Exhibit Early Metabolic Deficits and a Pathologically Low Leptin State Associated with Hypothalamic Dysfunction in Arcuate Neuropeptide Y Neurons

Lignende værker

• Amyloid-Beta Modulates Low-Threshold Activated Voltage-Gated L-Type Calcium Channels of Arcuate Neuropeptide Y Neurons Leading to Calcium Dysregulation and Hypothalamic Dysfunction
  af: Ishii, Makoto, et al.
  Udgivet: (2019)
• Increased Susceptibility to Ischemic Brain Damage in Transgenic Mice Overexpressing the Amyloid Precursor Protein
  af: Zhang, Fangyi, et al.
  Udgivet: (1997)
• Overexpression of Heparanase Lowers the Amyloid Burden in Amyloid-β Precursor Protein Transgenic Mice
  af: Jendresen, Charlotte B., et al.
  Udgivet: (2015)
• Ablation of the Leptin Receptor in the Hypothalamic Arcuate Nucleus Abrogates Leptin-Induced Sympathetic Activation
  af: Harlan, Shannon M., et al.
  Udgivet: (2011)
• Generation of Transgenic Cynomolgus Monkeys Overexpressing the Gene for Amyloid-β Precursor Protein
  af: Seita, Yasunari, et al.
  Udgivet: (2020)