Increased Susceptibility to Ischemic Brain Damage in Transgenic Mice Overexpressing the Amyloid Precursor Protein

Podobne zapisy

• Nox2-derived radicals contribute to neurovascular and behavioral dysfunction in mice overexpressing the amyloid precursor protein
  od: Park, Laibaik, i wsp.
  Wydane: (2008)
• Cyclo-Oxygenase-2 Gene Expression in Neurons Contributes to Ischemic Brain Damage
  od: Nogawa, Shigeru, i wsp.
  Wydane: (1997)
• Overexpression of Heparanase Lowers the Amyloid Burden in Amyloid-β Precursor Protein Transgenic Mice
  od: Jendresen, Charlotte B., i wsp.
  Wydane: (2015)
• Transgenic Mice Overexpressing Amyloid Precursor Protein Exhibit Early Metabolic Deficits and a Pathologically Low Leptin State Associated with Hypothalamic Dysfunction in Arcuate Neuropeptide Y Neurons
  od: Ishii, Makoto, i wsp.
  Wydane: (2014)
• Generation of Transgenic Cynomolgus Monkeys Overexpressing the Gene for Amyloid-β Precursor Protein
  od: Seita, Yasunari, i wsp.
  Wydane: (2020)