Multiple Sclerosis Autoantigen Myelin Basic Protein Escapes Control by Ubiquitination during Proteasomal Degradation

Lignende værker

• Glatiramer Acetate and Nanny Proteins Restrict Access of the Multiple Sclerosis Autoantigen Myelin Basic Protein to the 26S Proteasome
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  Udgivet: (2014)
• The ubiquitin-proteasome system and activation of NF-κB: involvement of the ubiquitin ligase KPC1 in p105 processing and tumor suppression
  af: Kravtsova-Ivantsiv, Yelena, et al.
  Udgivet: (2015)
• Ubiquitin-independent proteosomal degradation of myelin basic protein contributes to development of neurodegenerative autoimmunity
  af: Belogurov, Alexey, et al.
  Udgivet: (2015)
• Analysis of Myelin Basic Protein Fragmentation by Proteasome
  af: Bacheva, A. V., et al.
  Udgivet: (2009)
• The Transcriptome of Type I Murine Astrocytes under Interferon-Gamma Exposure and Remyelination Stimulus
  af: Kudriaeva, Anna, et al.
  Udgivet: (2017)