Hypoxia and Reoxygenation Induce Endothelial Nitric Oxide Synthase Uncoupling in Endothelial Cells through Tetrahydrobiopterin Depletion and S-Glutathionylation

समान संसाधन

• Integrated Redox Sensor and Effector Functions for Tetrahydrobiopterin- and Glutathionylation-dependent Endothelial Nitric-oxide Synthase Uncoupling
  द्वारा: Crabtree, Mark J., और अन्य
  प्रकाशित: (2013)
• Dexamethasone, tetrahydrobiopterin and uncoupling of endothelial nitric oxide synthase
  द्वारा: Tobias, Silke, और अन्य
  प्रकाशित: (2015)
• S-Glutathionylation Reshapes Our Understanding of Endothelial Nitric Oxide Synthase Uncoupling and Nitric Oxide/Reactive Oxygen Species-Mediated Signaling
  द्वारा: Zweier, Jay L., और अन्य
  प्रकाशित: (2011)
• Oxidation of tetrahydrobiopterin leads to uncoupling of endothelial cell nitric oxide synthase in hypertension
  द्वारा: Landmesser, Ulf, और अन्य
  प्रकाशित: (2003)
• Characterization of CD38 in the major cell types of the heart: endothelial cells highly express CD38 with activation by hypoxia-reoxygenation triggering NAD(P)H depletion
  द्वारा: Boslett, James, और अन्य
  प्रकाशित: (2018)