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Inhibition of calpain reduces oxidative stress and attenuates endothelial dysfunction in diabetes
AIMS: The present study was to investigate the role of calpain in reactive oxygen species (ROS) production in endothelial cells and endothelium-dependent vascular dysfunction under experimental conditions of diabetes. METHODS AND RESULTS: Exposure to high glucose activated calpain, induced apoptosis...
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| 主要な著者: | , , , , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
BioMed Central
2014
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| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4045988/ https://ncbi.nlm.nih.gov/pubmed/24886224 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1475-2840-13-88 |
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