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GLUT1 Deficiency in Cardiomyocytes Does not Accelerate the Transition from Compensated Hypertrophy to Heart Failure

AIMS: To determine whether endogenous GLUT1 induction and the increased glucose utilization that accompanies pressure overload hypertrophy (POH) are required to maintain cardiac function during hemodynamic stress, and to test the hypothesis that lack of GLUT1 will accelerate the transition to heart...

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Detalhes bibliográficos
Main Authors: Pereira, Renata O., Wende, Adam R., Olsen, Curtis, Soto, Jamie, Rawlings, Tenley, Zhu, Yi, Riehle, Christian, Abel, E. Dale
Formato: Artigo
Idioma:Inglês
Publicado em: 2014
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4037364/
https://ncbi.nlm.nih.gov/pubmed/24583251
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2014.02.011
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