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GLUT1 Deficiency in Cardiomyocytes Does not Accelerate the Transition from Compensated Hypertrophy to Heart Failure
AIMS: To determine whether endogenous GLUT1 induction and the increased glucose utilization that accompanies pressure overload hypertrophy (POH) are required to maintain cardiac function during hemodynamic stress, and to test the hypothesis that lack of GLUT1 will accelerate the transition to heart...
Αποθηκεύτηκε σε:
| Κύριοι συγγραφείς: | , , , , , , , |
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| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
2014
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4037364/ https://ncbi.nlm.nih.gov/pubmed/24583251 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2014.02.011 |
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