Inducible Overexpression of GLUT1 Prevents Mitochondrial Dysfunction and Attenuates Structural Remodeling in Pressure Overload but Does Not Prevent Left Ventricular Dysfunction

BACKGROUND: Increased glucose transporter 1 (GLUT1) expression and glucose utilization that accompany pressure overload‐induced hypertrophy (POH) are believed to be cardioprotective. Moreover, it has been shown that lifelong transgenic overexpression of GLUT1 in the heart prevents cardiac dysfunctio...

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Detalhes bibliográficos
Main Authors: Pereira, Renata O., Wende, Adam R., Olsen, Curtis, Soto, Jamie, Rawlings, Tenley, Zhu, Yi, Anderson, Steven M., Dale Abel, E.
Formato: Artigo
Idioma:Inglês
Publicado em: Blackwell Publishing Ltd 2013
Assuntos:
Original Research
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3835233/
https://ncbi.nlm.nih.gov/pubmed/24052497
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/JAHA.113.000301
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