Aggregation Modulators Interfere with Membrane Interactions of β(2)-Microglobulin Fibrils

Amyloid fibril accumulation is a pathological hallmark of several devastating disorders, including Alzheimer’s disease, prion diseases, type II diabetes, and others. Although the molecular factors responsible for amyloid pathologies have not been deciphered, interactions of misfolded proteins with c...

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Detalhes bibliográficos
Main Authors: Sheynis, Tania, Friediger, Anat, Xue, Wei-Feng, Hellewell, Andrew L., Tipping, Kevin W., Hewitt, Eric W., Radford, Sheena E., Jelinek, Raz
Formato: Artigo
Idioma:Inglês
Publicado em: The Biophysical Society 2013
Assuntos:
Proteins and Nucleic Acids
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3736666/
https://ncbi.nlm.nih.gov/pubmed/23931322
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bpj.2013.06.015
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