In vivo inactivation of erythrocyte S-adenosylhomocysteine hydrolase by 2'-deoxyadenosine in adenosine deaminase-deficient patients.

The cytotoxic nucleoside 2'-deoxyadenosine is excreted in excessive amounts by individuals with genetic deficiency of adenosine deaminase, and may be in part responsible for the severe combined immune dysfunction from which they suffer. Earlier studies from this laboratory showed that 2'-deoxyadenos...

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Detalhes bibliográficos

Publicado no:	J Clin Invest
Principais autores:	Hershfield, M S, Kredich, N M, Ownby, D R, Ownby, H, Buckley, R
Formato:	Artigo
Idioma:	Inglês
Publicado em:	American Society for Clinical Investigation 1979
Assuntos:	Research Article
Acesso em linha:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC372019/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/312296/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI109367
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