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Amyloid-β induced signaling by cellular prion protein and Fyn kinase in Alzheimer disease
Alzheimer disease (AD) is the most prevalent cause of dementia. Amyloid-β (Aβ) oligomers are potent synaptotoxins thought to mediate AD-related phenotypes. Cellular prion protein (PrP(C)) has been identified as a high-affinity receptor for Aβ oligomers. Herein, we review the functional consequences...
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| Główni autorzy: | , |
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| Format: | Artigo |
| Język: | Inglês |
| Wydane: |
Landes Bioscience
2013
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| Hasła przedmiotowe: | |
| Dostęp online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3609048/ https://ncbi.nlm.nih.gov/pubmed/22987042 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4161/pri.22212 |
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